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Found 2 translations.
| Name | Current message text |
|---|---|
| h English (en) | ===Stenosis and closure=== Over time, atheromata usually progress in size and thickness and induce the surrounding muscular central region (the media) of the [[artery]] to stretch out, which is termed ''remodeling''. Typically, remodeling occurs just enough to compensate for the atheroma's size such that the [[Caliber|calibre]] of the artery opening ([[Lumen (anatomy)|lumen]]) remains unchanged, until about 50% of the artery wall cross-sectional area consists of atheromatous tissue. [[File:Atheroma.jpg|thumb|302x302px|Narrowed arterial blood vessel blocked with an atheroma (artist's conception).]] If the muscular wall enlargement eventually fails to keep up with the enlargement of the atheroma volume, or a clot forms and organizes over the plaque, then the lumen of the artery becomes narrowed as a result of repeated ruptures, clots and fibrosis over the tissues separating the atheroma from the blood stream. This narrowing becomes more common after decades of living, increasingly more common after people are in their 30s to 40s. |
| h Japanese (ja) | ===狭窄と閉塞=== 時間の経過とともに粥腫の大きさと厚さは増大し、[[artery/ja|動脈]]の周囲の筋肉質な中心部（中膜）が伸展する。通常、リモデリングは粥腫の大きさを補うのに十分な程度に起こり、動脈開口部の[[Caliber/ja|口径]]（[[Lumen (anatomy)/ja|内腔]]）は変化しないが、動脈壁断面積の約50％が粥腫組織で構成されるようになる。 [[File:Atheroma.jpg|thumb|302x302px|粥腫で閉塞した狭窄動脈血管（想像図）。]] やがて筋壁の肥大がアテローム体積の肥大に追いつかなくなったり、プラーク上に血栓が形成されて組織化したりすると、アテロームと血流を隔てる組織の破裂、血栓、線維化が繰り返される結果、動脈の内腔が狭くなる。この狭窄は、何十年も生きているとより一般的になり、30代から40代になるとますます一般的になる。 |