Dihydrotachysterol: Difference between revisions

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Created page with "<languages /> <translate> {{Short description|Chemical compound}} {{Drugbox | verifiedrevid = 460787840 | IUPAC_name = (1''S'',3''E'',4''S'')-3-[(2''E'')-2-[(1''R'',3a''S'',7a''R'')-1-[(''E'',2''R'',5''R'')-5,6-Dimethylhept-3-en-2-yl]-7a-methyl-2,3,3a,5,6,7-hexahydro-1''H''-inden-4-ylidene]ethylidene]-4-methylcyclohexan-1-ol | image = Dihydrotachysterol.svg | alt = Structural formula | image2 = Dihydrotachysterol molecule spacefill.png | alt2 = Space-filling model of the..."
 
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'''Dihydrotachysterol''' (DHT) is a synthetic [[vitamin D]] analog activated in the [[liver]] that does not require [[renal]] hydroxylation like vitamin D<sub>2</sub> ([[ergocalciferol]]) and vitamin D<sub>3</sub> ([[cholecalciferol]]). DHT has a rapid onset of action (2 hours), a shorter half-life, and a greater effect on [[Mineralization (biology)|mineralization]] of bone salts than does vitamin D.
'''Dihydrotachysterol''' (DHT) is a synthetic [[vitamin D]] analog activated in the [[liver]] that does not require [[renal]] hydroxylation like vitamin D<sub>2</sub> ([[ergocalciferol]]) and vitamin D<sub>3</sub> ([[cholecalciferol]]). DHT has a rapid onset of action (2 hours), a shorter half-life, and a greater effect on [[Mineralization (biology)|mineralization]] of bone salts than does vitamin D.


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{{Sterols}}
{{Sterols}}
{{Vitamins}}
{{Vitamins}}
{{Vitamin D receptor modulators}}
{{Vitamin D receptor modulators}}


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{{二次利用|date=29 January 2023}}
{{二次利用|date=29 January 2023}}
[[Category:Secosteroids]]
[[Category:Secosteroids]]

Latest revision as of 22:28, 12 April 2024

Dihydrotachysterol
Structural formula
Space-filling model of the dihydrotachysterol molecule
Clinical data
AHFS/Drugs.comMicromedex Detailed Consumer Information
MedlinePlusa682335
ATC code
Identifiers
CAS Number
PubChem CID
DrugBank
ChemSpider
UNII
KEGG
ChEBI
Chemical and physical data
FormulaC28H46O
Molar mass398.675 g·mol−1
3D model (JSmol)
  (verify)

Dihydrotachysterol (DHT) is a synthetic vitamin D analog activated in the liver that does not require renal hydroxylation like vitamin D2 (ergocalciferol) and vitamin D3 (cholecalciferol). DHT has a rapid onset of action (2 hours), a shorter half-life, and a greater effect on mineralization of bone salts than does vitamin D.