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	<title>Translations:Obesity/35/en - Revision history</title>
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		<title>FuzzyBot: Importing a new version from external source</title>
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		<summary type="html">&lt;p&gt;Importing a new version from external source&lt;/p&gt;
&lt;p&gt;&lt;b&gt;New page&lt;/b&gt;&lt;/p&gt;&lt;div&gt;===Genetics===&lt;br /&gt;
{{Main|Genetics of obesity}}&lt;br /&gt;
[[File:La monstrua desnuda (1680), de Juan Carreño de Miranda..jpg|thumb|upright=1.3|alt=A painting of a dark haired pink cheeked obese nude young female leaning against a table. She is holding grapes and grape leaves in her left hand which cover her genitalia.|&amp;quot;La Monstrua Desnuda&amp;quot; (The Nude Monster), an 1680 painting by [[Juan Carreno de Miranda]] of a girl presumed to have [[Prader–Willi syndrome]]]]&lt;br /&gt;
Like many other medical conditions, obesity is the result of an interplay between genetic and environmental factors. [[Polymorphism (biology)|Polymorphisms]] in various [[gene]]s controlling [[appetite]] and [[metabolism]] predispose to obesity when sufficient food energy is present. As of 2006, more than 41 of these sites on the human genome have been linked to the development of obesity when a favorable environment is present. People with two copies of the [[FTO gene]] (fat mass and obesity associated gene) have been found on average to weigh 3–4&amp;amp;nbsp;kg more and have a 1.67-fold greater risk of obesity compared with those without the risk [[allele]]. The differences in BMI between people that are [[heritability|due to genetics]] varies depending on the population examined from 6% to 85%.&lt;/div&gt;</summary>
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