https://wiki.tiffa.net/w/index.php?action=history&feed=atom&title=Translations%3AInsulin_resistance%2F24%2FenTranslations:Insulin resistance/24/en - Revision history2026-09-14T22:38:21ZRevision history for this page on the wikiMediaWiki 1.43.0https://wiki.tiffa.net/w/index.php?title=Translations:Insulin_resistance/24/en&diff=126510&oldid=prevFuzzyBot: Importing a new version from external source2024-03-10T00:44:30Z<p>Importing a new version from external source</p>
<p><b>New page</b></p><div>In states of insulin resistance, [[beta cell]]s in the [[pancreas]] increase their production of insulin. This causes [[hyperinsulinemia|high blood insulin]] (hyperinsulinemia) to compensate for the high blood glucose. During this compensated phase of insulin resistance, [[Pancreatic beta cell function|beta cell function]] is upregulated, insulin levels are higher, and blood glucose levels are still maintained. If compensatory insulin secretion fails, then either fasting (impaired fasting glucose) or postprandial (impaired glucose tolerance) glucose concentrations increase. Eventually, type 2 diabetes occurs when glucose levels become higher as the resistance increases and compensatory insulin secretion fails. The inability of the β-cells to produce sufficient insulin in a condition of hyperglycemia is what characterizes the transition from insulin resistance to type 2 diabetes.</div>FuzzyBot