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	<title>Translations:Calcium channel blocker/30/en - Revision history</title>
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	<updated>2026-05-11T06:38:49Z</updated>
	<subtitle>Revision history for this page on the wiki</subtitle>
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		<title>FuzzyBot: Importing a new version from external source</title>
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		<updated>2023-11-07T09:46:48Z</updated>

		<summary type="html">&lt;p&gt;Importing a new version from external source&lt;/p&gt;
&lt;p&gt;&lt;b&gt;New page&lt;/b&gt;&lt;/p&gt;&lt;div&gt;Since blood pressure is in intimate feedback with cardiac output and peripheral resistance, with relatively low blood pressure, the [[afterload]] on the heart decreases; this decreases how hard the heart must work to eject blood into the aorta, so the amount of oxygen required by the heart decreases accordingly. This can help ameliorate symptoms of [[ischaemic heart disease]] such as [[angina pectoris]].&lt;br /&gt;
[[File:L-type D-subtype CaV1.3 calcium channel CACNA1D in human adrenal zona glomerulosa.jpg|thumb|right|250px|Immunohistochemical analysis of L-type calcium channel [[Cav1.3]] (CACNA1D) in human [[adrenal cortex]]: Marked immunoreactivity was detected in the [[zona glomerulosa]]. In the figure: ZG = zona glomerulosa, ZF = [[zona fasciculata]], AC = adrenal capsule. [[Immunohistochemistry]] was performed according to published methods.]]&lt;br /&gt;
Reducing the force of contraction of the myocardium is known as the negative [[inotropic]] effect of calcium channel blockers.&lt;/div&gt;</summary>
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