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	<title>Translations:Atheroma/5/en - Revision history</title>
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		<title>FuzzyBot: Importing a new version from external source</title>
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		<summary type="html">&lt;p&gt;Importing a new version from external source&lt;/p&gt;
&lt;p&gt;&lt;b&gt;New page&lt;/b&gt;&lt;/p&gt;&lt;div&gt;===Stenosis and closure===&lt;br /&gt;
Over time, atheromata usually progress in size and thickness and induce the surrounding muscular central region (the media) of the [[artery]] to stretch out, which is termed &amp;#039;&amp;#039;remodeling&amp;#039;&amp;#039;. Typically, remodeling occurs just enough to compensate for the atheroma&amp;#039;s size such that the [[Caliber|calibre]] of the artery opening ([[Lumen (anatomy)|lumen]]) remains unchanged, until about 50% of the artery wall cross-sectional area consists of atheromatous tissue.&lt;br /&gt;
[[File:Atheroma.jpg|thumb|302x302px|Narrowed arterial blood vessel blocked with an atheroma (artist&amp;#039;s conception).]]&lt;br /&gt;
If the muscular wall enlargement eventually fails to keep up with the enlargement of the atheroma volume, or a clot forms and organizes over the plaque, then the lumen of the artery becomes narrowed as a result of repeated ruptures, clots and fibrosis over the tissues separating the atheroma from the blood stream. This narrowing becomes more common after decades of living, increasingly more common after people are in their 30s to 40s.&lt;/div&gt;</summary>
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