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	<title>Translations:Atheroma/3/en - Revision history</title>
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	<updated>2026-11-02T13:43:13Z</updated>
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		<title>FuzzyBot: Importing a new version from external source</title>
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&lt;p&gt;&lt;b&gt;New page&lt;/b&gt;&lt;/p&gt;&lt;div&gt;==Signs and symptoms==&lt;br /&gt;
For most people, the first symptoms result from atheroma progression within the [[coronary arteries|heart arteries]], most commonly resulting in a [[myocardial infarction|heart attack]] and ensuing debility. The heart arteries are difficult to track because they are small (from about 5&amp;amp;nbsp;mm down to microscopic), they are hidden deep within the chest and they never stop moving.  Additionally, all mass-applied clinical strategies focus on both minimal cost and  the overall safety of the procedure. Therefore, existing diagnostic strategies for detecting atheroma and tracking response to treatment have been extremely limited. The methods most commonly relied upon, patient symptoms and [[cardiac stress testing]], do not detect any symptoms of the problem until atheromatous disease is very advanced because arteries enlarge, not constrict, in response to increasing atheroma. It is plaque ruptures, producing debris and clots which obstruct blood flow downstream, sometimes also locally (as seen on [[angiogram]]s), which reduce/stop blood flow. Yet these events occur suddenly and are not revealed in advance by either [[Cardiac stress test|stress tests]]&lt;/div&gt;</summary>
		<author><name>FuzzyBot</name></author>
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