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	<title>Translations:Angiotensin II receptor blocker/14/en - Revision history</title>
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	<updated>2026-11-10T07:58:08Z</updated>
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		<title>FuzzyBot: Importing a new version from external source</title>
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		<updated>2024-03-25T09:03:20Z</updated>

		<summary type="html">&lt;p&gt;Importing a new version from external source&lt;/p&gt;
&lt;p&gt;&lt;b&gt;New page&lt;/b&gt;&lt;/p&gt;&lt;div&gt;Indeed, as a consequence of AT&amp;lt;sub&amp;gt;1&amp;lt;/sub&amp;gt; blockade, ARBs increase angiotensin II levels several-fold above baseline by uncoupling a [[negative-feedback]] loop. Increased levels of circulating angiotensin II result in unopposed stimulation of the AT&amp;lt;sub&amp;gt;2&amp;lt;/sub&amp;gt; receptors, which are, in addition, upregulated. However, recent data suggest AT&amp;lt;sub&amp;gt;2&amp;lt;/sub&amp;gt; receptor stimulation may be less beneficial than previously proposed, and may even be harmful under certain circumstances through mediation of growth promotion, [[fibrosis]], and [[hypertrophy]], as well as eliciting [[atherogenesis|proatherogenic]] and [[proinflammatory]] effects.&lt;/div&gt;</summary>
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